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Pulmonary embolism triggered by cold agglutinin syndrome in mycoplasma pneumoniae pneumonia
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  • Zeyu Wang,
  • Ni Yang,
  • Wenliang Song,
  • Bo Wu,
  • Jingli Yan,
  • Wei Xu
Zeyu Wang
Shengjing Hospital of China Medical University

Corresponding Author:[email protected]

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Ni Yang
Shengjing Hospital of China Medical University
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Wenliang Song
Shengjing Hospital of China Medical University
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Bo Wu
Shengjing Hospital of China Medical University
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Jingli Yan
Shengjing Hospital of China Medical University
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Wei Xu
Shengjing Hospital of China Medical University
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Abstract

Mycoplasma (M.) pneumoniae is a common pathogen causing respiratory infections in children. Pulmonary embolism is a rare complication that may be life-threatening if not diagnosed early and treated promptly. Here, we report the case of an 11-year-old patient with pulmonary embolism associated with M. pneumoniae pneumonia. The patient developed uncorrectable hypoxemia and received venovenous extracorporeal membrane oxygenation treatment. Although the mechanism of thrombosis after M. pneumoniae infection remains unknown, an increase in the cold agglutinin titer indicates that cold agglutinin syndrome might be the mechanism of this pathological change. We thought that patients who have positive cold agglutinin after M. pneumoniae infection should be monitored for the possibility of thrombosis formation.