All stresses induced a dose-dependent decline in relative growth rate (RGR), confirming that the plants perceived and responded to the stresses in a dose-dependent manner (Fig. 2a). By contrast, seed production showed a different response to the stresses. The lowest levels of disease by Pst and Pc stimulated seed production, whereas the highest stress levels by these diseases had no statistically significant effect on seed production (Fig. 2b). This suggests that Arabidopsis can adapt to both diseases by compensating the reduced growth during pathogen exposure with increased seed production at the end of its life cycle. Conversely, increasing levels of soil salinity caused a dose-dependent reduction in seed production (Fig. 2b), indicating that Arabidopsis does not recover as efficiently from this stress as it does from disease by Pst or Pc. Similar patterns were observed for seed viability, where Pst and Pc failed to have an effect (Fig. 2c and Fig. S1a,b), whereas soil salinity caused a dramatic dose-dependent decline in seed viability (Fig. 2c and Fig. S1c), which was absent in F2 seeds after one stress-free F1 generation (Fig. S1d).