Supporting information for Figures
Figure. S1. The transcriptional response of members of pepper
NAC family and CaNAC2c to HTS or RSI.
Figure. S2. Deduced amino acid sequences of pepper NAC2
proteins and their phylogenic relationship.
Figure. S3. The promoter activity of pCaNAC2c and its
series 5’ deletions to exogenous application of MeJA, ABA, IAA and GA3.
Figure. S4. Nuclear localization of the CaNAC2c in N.
benthamiana epidermal cells.
Figure.S5 . The CaNAC2c overexpressing N. benthamianaplants inhibited growth and retarded development compared to the wild
type control plants.
Figure. S6. The CaNAC2c overexpressing N.
benthamiana plants exhibited enhanced Fv/Fm, △F/Fm and decreased ion
leakage and lower levels of DAB or NBT staining upon HTS.
Figure. S7. CaHSFA5 acts as negative regulator in pepper
growth.
Figure. S8. The silencing of CaHSFA5 significantly
promoted the growth of CaNAC2c overexpressing N.
benthamiana plants.
Figure. S9. The silencing of NbHSFA5 significantly
decreased thermotolerance but not affect the resistance ofCaNAC2c overexpressing N. benthamiana plants to RSI.
Figure. S10. The silencing of NtHSFA5 did not affect
transcription of NbDEF1 but significantly downregulatedNtsHSP , NtAPX in CaNAC2c overexpressing N.
benthamiana plants.